Chemokines in the pathogenesis of vascular disease.

By I. Charo, M. Taubman. Circulation Research, 2004

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Social Support and Health: A Review of Physiological Processes Potentially Underlying Links to Disease Outcomes (2006, background): “Monocytes and T-lymphocytes are then recruited to sites of inflammation and migrate into vessel walls via various cytokines/chemokines (e.g., monocyte chemoattractant protein-1) that are released from vascular cell walls (Charo and Taubman, 2004).”
Flavonoids as anti-inflammatory agents: implications in cancer and cardiovascular disease (2009, background): “Transendothelial migration of leukocytes during the inflammatory process is triggered by chemotactic proteins such as monocyte chemoattractant protein-1 (MCP-1) [165] as well as by proinflammatory cytokines secreted by macrophages and T cells, such as TNF-a, IL-1, IL-6, [166, 167] and growth factors”
Monocytes in atherosclerosis: subsets and functions (2010, background): “Chemokine receptors Chemokines and their receptors have been shown to be important for leukocyte trafficking and migration related to atherosclerosis”
Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2 (2006, background): “Blocking CCR2–MCP-1 interactions has been suggested as a potential therapeutic approac”