The emergence and evolution of methicillin-resistant Staphylococcus aureus.
By K. Hiramatsu, L. Cui, M. Kuroda, Teruyo Ito. Trends in Microbiology, 2001
869 citations recorded by the source corpus.
Subfields: Clinical Biochemistry, Infectious Diseases, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/3975de2caf7fdaf22170347b0d299c6d44a3adfa
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Clinical Biochemistry
In Infectious Diseases
- Hermínia de Lencastre
- F. Lowy
- Duarte C. Oliveira
- K. Hiramatsu
- Teruyo Ito
- Robert S. Daum
- K. Okuma
- Michael Z. David
In Molecular Biology
- Hermínia de Lencastre
- Duarte C. Oliveira
- F. Lowy
- K. Hiramatsu
- Teruyo Ito
- Robert S. Daum
- C. Tiensasitorn
- K. Okuma
Citations in the corpus
- Antimicrobial resistance: the example of Staphylococcus aureus. (2003, background): “To date, four different SCCmec elements varying in size from 21 to 67 kb have been characterized (33).”
- Penicillin-binding proteins and beta-lactam resistance. (2008, background): “A detailed presentation of these genetic elements can be found elsewhere (Hiramatsu et al., 2001; Ender et al., 2004).”
- Pathogenesis of methicillin-resistant Staphylococcus aureus infection. (2008, background): “The initial reservoir of SCCmec is unclear but may have been a coagulase-negative staphylococcal species [46–48].”
- The molecular evolution of methicillin-resistant Staphylococcus aureus. (2007, neutral): “1) [4,8,9,11,12].”
- Dissemination of New Methicillin-Resistant Staphylococcus aureus Clones in the Community (2002, background): “This raised a possibility that CMRSA might have an origin of derivation distinct from that of H-MRSA, and type-IV SCCmec could be its unique genetic marker (14).”
- Community-Associated Methicillin-Resistant Staphylococcus aureus: Epidemiology and Clinical Consequences of an Emerging Epidemic (2010, background): “additional mec complexes, called classes C and D, have also been identified (390), although the class D complex has been found only in coagulase-negative Staphylococcus species.”
- The evolution of Staphylococcus aureus. (2008, neutral): “1) (Hiramatsu et al., 2001; Katayama et al., 2001; Oliveira et al., 2001, 2006a; Daum et al., 2002; Ito et al., 2004).”
- Multiplex PCR Strategy for Rapid Identification of Structural Types and Variants of the mec Element in Methicillin-Resistant Staphylococcus aureus (2002, background): “One of the conclusions emerging from these studies was that a complete characterization of MRSA lineages requires not only identification of the genetic background of the bacteria but also identification of the structural types of the large and heterologous mec element, which carries methicillin res”
- Action and resistance mechanisms of antibiotics: A guide for clinicians (2017, background): “aureus that contains resistance gene mec A.[4,15,16] mec A gene encodes PBP2a protein, a new penicillin-binding protein, that is required to change a native staphylococcal PBP.”
- Pulsed-Field Gel Electrophoresis Typing of Oxacillin-Resistant Staphylococcus aureus Isolates from the United States: Establishing a National Database (2003, methodology): “The nomenclature chosen is similar to that described by Enright et al. (20), Feil et al. (21), and Hiramatsu et al. ( 29 ), except that the genetic backgrounds of the strains in this study were determined by SmaI PFGE and designated as PFTs.”
- Functional Gold Nanoparticles as Potent Antimicrobial Agents against Multi-Drug-Resistant Bacteria (2014, background): “aureus has been a stumbling block for antimicrobial treatment, overcoming most of the therapeutic chemo-agents developed in the past five decades.(29) Particularly, MRSA has emerged as “superbug”, resistant to most antibiotics commonly used for the staph infections.”
- Community-acquired methicillin-resistant Staphylococcus aureus: a meta-analysis of prevalence and risk factors. (2003, background): “resistance among staphylococci by acquisition of the mecA gene does not occur in the community, even though molecular epidemiological studies have suggested that this occurs only very rarely in any setting [69, 70].”