Inflammation, Atherosclerosis and Coronary Artery Disease

By Y. Momiyama, H. Adachi, D. Fairweather, N. Ishizaka, Emi Saita. Clinical Medicine Insights: Cardiology, 2014

3,021 citations recorded by the source corpus.

Article types include original clinical and basic research articles, case reports, commentaries, meeting reports, methodology, perspectives. Research into inflammation, atherosclerosis and coronary artery disease, including observational studies, clinical trials, epidemiology, and advances in applied (translational) research. The supplement is intended to include overviews of new concepts in pathophysiology, natural history, diagnostic strategies, and treatment approaches.

Subfields: Cardiology and Cardiovascular Medicine, Epidemiology, Immunology

View Paper: https://www.semanticscholar.org/paper/2be15c9a4f07e09d12b295366a6d62f632078dcb

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Citations in the corpus

European Guidelines on Cardiovascular Disease Prevention in Clinical Practice (Version 2012) (2012, neutral): “Atherosclerosis is an inflammatory disease in which immune mechanisms interact with metabolic risk factors to initiate, propagate, and activate lesions in the arterial tree.(170) Several diseases in which infection or non-infectious inflammatory processes determine the clinical picture are associate”
Oral microbiomes: more and more importance in oral cavity and whole body (2018, background): “It can thus be considered both a metabolic and an inflammatory disease (Hansson, 2005; Koren et al., 2011).”
Inflammatory cytokines and risk of coronary heart disease: new prospective study and updated meta-analysis. (2014, neutral): “Anal Biochem 2005 Mar 15;338(2):294-298.”
Oxidative stress and reactive oxygen species in endothelial dysfunction associated with cardiovascular and metabolic diseases. (2018, background): “The recruitment of LDL cholesterol into the arterial wall also leads to activation of the NLRP3 inflammasome, since excess cholesterol is partially deposited as cholesterol crystals [157-158], which are fully internalized by human macrophages by phagocytosis and promote IL-1β and IL-18 generation [1”
Elevated Remnant Cholesterol Causes Both Low-Grade Inflammation and Ischemic Heart Disease, Whereas Elevated Low-Density Lipoprotein Cholesterol Causes Ischemic Heart Disease Without Inflammation (2013, background): “The causal association of nonfasting remnant cholesterol with low-grade inflammation observed in this study could be explained by remnant cholesterol causing atherosclerosis, a disease with a known inflammatory element.(14) Randomized, clinical trials have found that statin therapy prevents cardiova”
Association between plasma ADAMTS-7 levels and ventricular remodeling in patients with acute myocardial infarction (2015, neutral): “ADAMTS-7 is involved in the proliferative response to vascular injury in a way that is similar to the progressive phase of atherosclerosis [17].”
Low-grade inflammation, diet composition and health: current research evidence and its translation (2015, neutral): “ation, and (4) inducing plaque instability and eventual rupture((8,9)).”
National Lipid Association Recommendations for Patient-Centered Management of Dyslipidemia: Part 2. (2015, background): “Role of inflammation in the pathogenesis of atherosclerosis in HIV-infected patients Atherosclerosis is a chronic inflammatory process in which plaque formation is triggered by arterial wall injury, lipoprotein deposition, endothelial activation, and proinflammatory molecules.(633) An atheroscleroti”
Endothelial Cell Dysfunction and the Pathobiology of Atherosclerosis (2016, neutral): “One important mechanistic link seems to be the capacity of lipid-laden, dysfunctional adipocytes to generate a variety of proinflammatory adipokines (eg, tumor necrosis factor), which can act locally within adipose tissue to activate macrophages and microvascular endothelium and also systemically to”
Endothelial dysfunction and vascular disease – a 30th anniversary update (2017, background): “…of their dysfunction, the endothelial cells cannot produce enough NO in response to platelets and thrombin and this NO deficiency permits the inflammatory reaction leading to atherosclerosis (Ross 1999, Aikawa & Libby 2004, Hansson 2005, Barton et al. 2007, Vanhoutte 2009b, Vanhoutte et al. 2009).”
Deep Survival Analysis (2016, background): “It is the leading cause of death worldwide (Hansson, 2005; Pagidipati and Gaziano, 2013).”
Damage-Associated Molecular Patterns in Inflammatory Diseases (2018, background): “Atherosclerosis is an inflammatory disease of the arterial wall, in which the vessels narrow due to accumulating plaques of inflammatory cells and lipids (59).”
NLRP3 inflammasome: From a danger signal sensor to a regulatory node of oxidative stress and inflammatory diseases (2015, neutral): “…. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 302 References . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 304”
Update on acute coronary syndromes: the pathologists' view. (2013, background): “Atherosclerosis is an innate inflammatory disease in which smoldering inflammatory activity is not confined to just a few atherosclerotic lesions but is present, more or less, in all such lesions throughout the body.(86,87) In contrast, vulnerable plaques are relatively rare,(76) and inflammation ma”