Endothelial Cell Dysfunction and the Pathobiology of Atherosclerosis
By Peter Libby, Karin E. Bornfeldt, A. Tall. 2016
1,766 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/e5f23e50546a5260e5785c4735cc3fd471cad29b
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Citations in the corpus
- Activation of Nrf2/HO-1 signaling: An important molecular mechanism of herbal medicine in the treatment of atherosclerosis via the protection of vascular endothelial cells from oxidative stress (2021, background): “However, despite continued progress in research on various aspects of natural agents exerting protective effects against EC injury by activating Nrf2/HO-1 signaling, the development of new drugs for the treatment of atherosclerosis (AS) and other CVDs based on these agents will require more detailed”
- COVID-19 is, in the end, an endothelial disease (2020, background): “Positioned at the key interface between the blood and tissues, the endothelium normally resists prolonged contact with the leucocytes that abound in blood that bathes the intimal surface.(3,24) Stationed as the sentinel, the endothelium serves as the portal governing the entry”