Elevated Remnant Cholesterol Causes Both Low-Grade Inflammation and Ischemic Heart Disease, Whereas Elevated Low-Density Lipoprotein Cholesterol Causes Ischemic Heart Disease Without Inflammation
By A. Varbo, M. Benn, A. Tybjærg-Hansen, Børge G. Nordestgaard. Circulation, 2013
604 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Epidemiology, Surgery
View Paper: https://www.semanticscholar.org/paper/4034d9a6b73ff6ff3c9c991ff44c866b46033fa7
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Endocrinology, Diabetes and Metabolism
- Børge G. Nordestgaard
- Jan Boren
- Marja-Riitta Taskinen
- M. Chapman
- Alberico L. Catapano
- L. Tokgözoğlu
- Bart Staels
- I. Graham
In Epidemiology
In Surgery
- Børge G. Nordestgaard
- Jan Boren
- M. Chapman
- Marja-Riitta Taskinen
- Alberico L. Catapano
- L. Tokgözoğlu
- Bart Staels
- S. Kersten
Citations in the corpus
- Triglyceride-rich lipoproteins and their remnants: metabolic insights, role in atherosclerotic cardiovascular disease, and emerging therapeutic strategies—a consensus statement from the European Atherosclerosis Society (2021, positive): “This point is especially pertinent given that PESA reported the presence of peripheral atherosclerotic plaques in 58% of middle-aged individuals with elevated TG levels (>_1.7 mmol/L), low-to-moderate cardiovascular risk, and high or normal LDL-C.”
- Triglyceride-Rich Lipoproteins and Atherosclerotic Cardiovascular Disease: New Insights From Epidemiology, Genetics, and Biology. (2016, neutral): “A necessity for triglyceride-rich lipoproteins to cause atherosclerosis and ASCVD is their ability to enter into the arterial intima, the location of the atherosclerotic plaque, leading ultimately to ASCVD.”