Regulation of cancer cell migration and bone metastasis by RANKL
By D. H. Jones, T. Nakashima, Otto Sanchez, I. Kozieradzki, S. Komarova, I. Sarosi, S. Morony, E. Rubin, R. Sarao, C. Hojilla and 8 more. Nature, 2006
783 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/1777d6c3ecb00174fffdd032222dd71f89cd876a
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Citations in the corpus
- Genetics and biology of prostate cancer (2018, background): “Activated RANK–RANKL signaling in prostate cancer cells is also implicated in the colonization of cancer cells in the bone (Jones et al. 2006).”
- Cancer metastasis: building a framework. (2006, background): “A recent study suggests that this receptor-ligand combination may predispose breast-cancer cells to bone colonization (Jones et al., 2006).”
- Receptor activator of nuclear factor kappaB ligand and osteoprotegerin regulation of bone remodeling in health and disease. (2008, unknown): “cer cells have been shown to express RANK (101, 336), which”
- CD44+/CD24- breast cancer cells exhibit enhanced invasive properties: an early step necessary for metastasis (2006, background): “A recent study described the role of the receptor activator of nuclear factor-κB (RANK)/ RANK ligand in metastasis of breast cancer cells to bone [16].”
- Mechanistic Pathways of Sex Differences in Cardiovascular Disease. (2017, background): “The receptor activator of NF- B ligand (RANKL), its receptor RANK, and the decoy receptor osteoprotegerin also appear to be involved in the inflammatory process (146, 176, 350).”
- Extracellular matrix: a dynamic niche component during cancer progression (2012, unknown): “tumor cells to the lung or bone marrow (Jones et al., 2006).”