Inflammation 2010: new adventures of an old flame.

By R. Medzhitov. Cell, 2010

1,641 citations recorded by the source corpus.

Subfields: Epidemiology, Molecular Biology, Nutrition and Dietetics

View Paper: https://www.semanticscholar.org/paper/166d91d1dbbaf4a13c0660e59a0b900fee9778d1

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Citations in the corpus

Stress, Inflammation, and Defense of Homeostasis (2014, background): “These sensory cells detect noxious insults (pathogens, toxins, irritants, etc.) either directly (e.g., through PRRs) or indirectly through their effect on tissue homeostasis (e.g., by sensing unscheduled cell death, ECM degradation products, tissue damage, etc.) (Medzhitov, 2010).”
Macrophages: development and tissue specialization. (2015, background): “It is also a response to physiological noxious conditions associated with tissue remodeling, as well as mechanical stress (2, 3).”
The Role of the Transcription Factor CREB in Immune Function (2010, background): “In particular, TLRs are PRRs that mediate cellular responses to lipopeptides (TLRs 1, 2, and 6), LPS (TLR4), flagellum (TLR5), and microbial RNA and DNA nucleotide sequences (TLRs 3, 7, 8, and 9) (11, 12).”
Damage-Associated Molecular Patterns in Inflammatory Diseases (2018, background): “Although inflammation is a protective response to eliminate harmful stimuli, initiate tissue repair, and restore health, it can also contribute to the development of various diseases, such as autoimmune diseases, cardiovascular diseases, and neurodegenerative diseases, if it is not properly regulate”
Inflammatory responses and inflammation-associated diseases in organs (2015, background): “Inflammation is the immune system’s response to harmful stimuli, such as pathogens, damaged cells, toxic compounds, or irradiation [1], and acts by removing injurious stimuli and initiating the healing process [2].”
The role of oxidative stress during inflammatory processes (2013, background): “…molecular patterns (PAMPs), activate innate immune defenses through the activation of the so-called pattern recognition receptors (PRRs) and downstream cellular pro-inflammatory signaling, with the ultimate goal of eradicating the pathogen (Barton, 2008; Kono and Rock, 2008; Medzhitov, 2010b).”
Sepsis: current dogma and new perspectives. (2014, background): “…of resolvins that accelerate the efflux of inflammatory cells and debris, and feedback from the nervous system that suppresses the release of inflammatory mediators and stimulates the release of resolvins (Nathan and Ding, 2010; Schwab et al., 2007; Medzhitov, 2010; Andersson and Tracey, 2012a).”
microRNA regulation of inflammatory responses. (2012, background): “The often distinct clinical manifestations of chronic inflammation have allowed recognition of this process in humans for thousands of years (1).”
Role of Antioxidants and Natural Products in Inflammation (2016, background): “Classical pathway of complement is initiated by producing antibodies by natural antibodies, C-reactive protein, or serum amyloid protein [5].”