Ablation of Insulin-Producing Neurons in Flies: Growth and Diabetic Phenotypes

By Eric J. Rulifson, S. K. Kim, R. Nusse. Science, 2002

1,078 citations recorded by the source corpus.

Subfields: Aging, Cellular and Molecular Neuroscience, Ecology

View Paper: https://www.semanticscholar.org/paper/11f69b81594d4f28554f13fdc3b1377788710a96

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Aging

1. Jae H. Park
2. Gyunghee G. Lee

In Cellular and Molecular Neuroscience

1. Jae H. Park
2. Gyunghee G. Lee
3. R. Tjian
4. D. Kodrík

In Ecology

1. Jae H. Park
2. Gyunghee G. Lee
3. D. Kodrík

Citations in the corpus

• A Single-Cell Transcriptome Atlas of the Aging Drosophila Brain (2018, neutral): “…(2018); Doherty et al. (2009); Goto et al. (2001); Klarsfeld et al. (2004); Komiyama et al. (2003); Kunst et al. (2014); Luan et al. (2006); Medioni et al. (2014); Melcher and Pankratz (2005); Min et al. (2016); Park et al. (2000); Park et al. (2008); Rulifson et al. (2002); Spindler et al. (2009).”
• A nutrient sensor mechanism controls Drosophila growth. (2003, background): “Taken together, these results demonstrate that activa-and a dilp2-GAL4 driver (Rulifson et al., 2002) were used to selectively induce slifAnti in the seven dilp-expressing tion of the FB amino acid sensor does not induce a modulation of neuronal dilp expression.”
• Hemolymph Sugar Homeostasis and Starvation-Induced Hyperactivity Affected by Genetic Manipulations of the Adipokinetic Hormone-Encoding Gene in Drosophila melanogaster (2004, methodology): “The supernatant was transferred to a fresh tube and an aliquot (2 l) was applied for trehalose or glucose assays using a commercial kit (17-25; Sigma, St. Louis) as described in Rulifson et al. (2002).”
• Control of cell number by Drosophila FOXO: downstream and feedback regulation of the insulin receptor pathway. (2003, background): “2002) and that food limitation decreases the Drosophila insulin-like peptide (DILP) levels (Ikeya et al. 2002; Rulifson et al. 2002).”
• Insulin receptor isoforms and insulin receptor/insulin-like growth factor receptor hybrids in physiology and disease. (2009, neutral): “a development delay and growth retardation (153).”