Mechanisms underlying differential responses to FGF signaling.
By L. Dailey, D. Ambrosetti, A. Mansukhani, C. Basilico. Cytokine & growth factor reviews, 2005
690 citations recorded by the source corpus.
Subfields: Molecular Biology
View Paper: https://www.semanticscholar.org/paper/11a9a912cb0c0ef421d66db13d65bb03a8a8f1b2
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Citations in the corpus
- Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale. (2010, neutral): “phosphatidylinositol 3-kinase (PI3K)/Akt pathway (85).”
- Vascular endothelial growth factor (VEGF) signaling in tumor progression. (2007, background): “[259] Dailey L, Ambrosetti D, Mansukhani A, Basilico C.”
- The FGF family: biology, pathophysiology and therapy (2009, neutral): “Phosphorylation of FRS2 is essential for activation of the Ras–mitogen-activated protein kinase (MAPK) and phosphoinositide 3-kinase–Akt signalling pathway”
- Fibroblast Growth Factors: Biology, Function, and Application for Tissue Regeneration (2010, background): “Formation of the FRS2 signaling complex results in activation of RAS/MAP kinase [25] as well as PI3 kinase/AKT pathways [26].”
- Fibroblast growth factor signalling: from development to cancer (2010, background): “The downstream effect of MAPK signalling is mostly cell proliferation; for example, through the induction of cyclin D1 expression, thereby promoting entry into the S phase of the cell cycl”