Hypersensitive response-related death

By M. C. Heath. Plant Molecular Biology, 2000

1,048 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/027679ac37bc590ce388c743c1dea77f43e7741c

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Citations in the corpus

Coffee resistance to the main diseases: leaf rust and coffee berry disease (2006, background): “…(Grant and Mansfield 1999; Guest et al., 1999; Heath, 2000a, Dangl and Jones, 2001; Peck, 2003), involvement of plasma-membrane-bound NADPH oxidases, cell-wall-bound peroxidases, amino oxidases in the apoplast and phosphoproteins (Bestwick et al., 1997; Heath, 2000a, Laloi et al., 2004).”
Betacyanin synthesis in red beet (Beta vulgaris) leaves induced by wounding and bacterial infiltration is preceded by an oxidative burst (2004, background): “HR is a form of programmed cell death at the infection site that restricts pathogen growth and spread in the plant [18].”
A Dual Role for Microbial Pathogen-Derived Effector Proteins in Plant Disease and Resistance (2002, background): “The HR is a form of programmed cell death occurring at the site of infection (Heath, 2000; Morel and Dangl, 1997).”
Defence responses induced by potassium phosphonate in Phytophthora palmivora-challenged Arabidopsis thaliana (2005, background): “Successful disease resistance is rarely the consequence of a single host response, but results from a series of coordinated events in time and space [15].”
Elicitors, effectors, and R genes: the new paradigm and a lifetime supply of questions. (2007, background): “The hypersensitive response (HR) is a robust defense response frequently associated with R genemediated resistance, and includes the death of plant cells local to the site of infection (75).”
Genome-Wide Analysis of NBS-LRR–Encoding Genes in Arabidopsis Online version contains Web-only data. Article, publication date, and citation information can be found at www.plantcell.org/cgi/doi/10.1105/tpc.009308. (2003, background): “The plant resistance response triggered by R gene recognition also includes increased expression of defense genes, generation of reactive oxygen species, production or release of salicylic acid, ion fluxes, and other factors (Heath, 2000).”
The role and regulation of programmed cell death in plant–pathogen interactions (2004, methodology): “The reader may also find several older reviews useful for insight into this topic (Greenberg, 1997; Morel and Dangl, 1997; Heath, 2000).”
Mitochondrial reactive oxygen species in cell death signaling. (2002, background): “[90] M.”
The antioxidant systems vis-à-vis reactive oxygen species during plant–pathogen interaction (2003, background): “Apart from the oxidative burst, HR is characterised by other metabolic disturbances, such as ion flux across the plasma membrane (Ca influx and K, Cl efflux) as well as changes in pH and plasma membrane depolarisation [45].”
Recent Molecular and Genomic Studies on Stress Tolerance of Forage and Turf Grasses (2006, background): “The hypersensitive response is a rapid reaction, which usually occurs within 24 h of pathogen attack and leads either directly or indirectly to localized cell death, preventing further spread of the pathogen (Heath, 2000).”
Induction and modulation of resistance in tomato plants against Fusarium wilt disease by bioagent fungi (arbuscular mycorrhiza) and/or hormonal elicitors (Jasmonic acid & Salicylic acid): 2-Changes in the antioxidant enzymes, phenolic compounds and pathogen related- proteins. (2007, background): “phytoalexins (Heath 2000; Agrios 2005); activation and/or synthesis of defense peptides and proteins (Castro”
Inducible direct plant defense against insect herbivores: A review (2008, background): “HR involves the rapid death of cells at the infection site, which prevents further spread of the pathogen, causing it to starve to death (Heath, 2000).”