D. Ditsworth
Molecular Biology · 2004–2008
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Molecular Biology — 24 citations into it; #16,705 of 17,773 by trust here, #6,685 by citations
Whom D. Ditsworth trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts D. Ditsworth overall
Papers from D. Ditsworth's perspective
Ordered by how strongly the researchers D. Ditsworth trusts endorse each paper. Same corpus, a different order for every researcher.
- The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues. (Molecules and Cells)
- BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis. (Molecules and Cells)
- BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak. (Genes & Development)
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (Science)
- Live or let die: the cell's response to p53 (Nature Reviews. Cancer)
- Proliferation, cell cycle and apoptosis in cancer (Nature)
- BAX and BAK Regulation of Endoplasmic Reticulum Ca2+: A Control Point for Apoptosis (Science)
- The role of p53 in determining sensitivity to radiotherapy (Nature Reviews. Cancer)
- Bax and Bak can localize to the endoplasmic reticulum to initiate apoptosis (Journal of Cell Biology)
- Mediation of Poly(ADP-Ribose) Polymerase-1-Dependent Cell Death by Apoptosis-Inducing Factor (Science)
Their papers
- Brick by brick: metabolism and tumor cell growth. (2008) — 1,028 citations
- Alkylating DNA damage stimulates a regulated form of necrotic cell death. (2004) — 677 citations