Shuo-Chien Ling

Neurology · 2011–2021

A researcher in the Science corpus: 4 papers; trusts 4 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom Shuo-Chien Ling trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. J. Buchan
  2. Roy Parker
  3. Oliver D. King
  4. M. Carrasco
  5. M. Polymenidou
  6. L. Igaz

Papers from Shuo-Chien Ling's perspective

Ordered by how strongly the researchers Shuo-Chien Ling trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis (Science)
  2. TDP-43 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis (Science)
  3. TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis. (Biochemical and Biophysical Research Communications - BBRC)
  4. Disturbance of Nuclear and Cytoplasmic TAR DNA-binding Protein (TDP-43) Induces Disease-like Redistribution, Sequestration, and Aggregate Formation* (Journal of Biological Chemistry)
  5. Structural determinants of the cellular localization and shuttling of TDP-43 (Journal of Cell Science)
  6. Mutations in the FUS/TLS Gene on Chromosome 16 Cause Familial Amyotrophic Lateral Sclerosis (Science)
  7. Cell-Free Formation of RNA Granules: Low Complexity Sequence Domains Form Dynamic Fibers Within Hydrogels (Cell)
  8. Pathological TDP‐43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations (Annals of Neurology)
  9. Null mutations in progranulin cause ubiquitin-positive frontotemporal dementia linked to chromosome 17q21 (Nature)
  10. Expanded GGGGCC hexanucleotide repeat in non-coding region of C9ORF72 causes chromosome 9p-linked frontotemporal dementia and amyotrophic lateral sclerosis (Neuron)

Their papers