R. Schnellmann
Molecular Biology · 2017
A researcher in the Science corpus: 1 paper; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 50 citations into it
- Physiology — 22 citations into it
- Clinical Biochemistry — 9 citations into it
Whom R. Schnellmann trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- R. Scarpulla (in Physiology)
- D. C. Chan (in Molecular Biology)
Papers from R. Schnellmann's perspective
Ordered by how strongly the researchers R. Schnellmann trusts endorse each paper. Same corpus, a different order for every researcher.
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
- Disruption of Fusion Results in Mitochondrial Heterogeneity and Dysfunction* (Journal of Biological Chemistry)
- Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
- Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
- PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
- Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin (Proceedings of the National Academy of Sciences of the United States of America)
- Mitochondrial rhomboid PARL regulates cytochrome c release during apoptosis via OPA1-dependent cristae remodeling. (Cell)
- Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission* (Journal of Biological Chemistry)
Their papers
- Mitochondrial energetics in the kidney (2017) — 1,186 citations