Sarah R Pickles
Molecular Biology · 2018
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 43 citations into it
- Epidemiology — 40 citations into it
Whom Sarah R Pickles trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Sarah R Pickles overall
Papers from Sarah R Pickles's perspective
Ordered by how strongly the researchers Sarah R Pickles trusts endorse each paper. Same corpus, a different order for every researcher.
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
- Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
- Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
- The mitochondrial deubiquitinase USP30 opposes parkin-mediated mitophagy (Nature)
- p62/SQSTM1 Binds Directly to Atg8/LC3 to Facilitate Degradation of Ubiquitinated Protein Aggregates by Autophagy* (Journal of Biological Chemistry)
- PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Journal of Cell Biology)
- Broad activation of the ubiquitin–proteasome system by Parkin is critical for mitophagy (Human Molecular Genetics)
- PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- The art of mitochondrial maintenance (2018) — 1,754 citations