Eli A. Stahl

Genetics · 2010–2022

A researcher in the Science corpus: 22 papers; trusts 10 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Genetics — 563 citations into it

Whom Eli A. Stahl trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

Who trusts Eli A. Stahl overall

Papers from Eli A. Stahl's perspective

Ordered by how strongly the researchers Eli A. Stahl trusts endorse each paper. Same corpus, a different order for every researcher.

Principal components analysis corrects for stratification in genome-wide association studies
Common SNPs explain a large proportion of heritability for human height (Nature Genetics)
Genotype Imputation with Thousands of Genomes (G3: Genes, Genomes, Genetics)
A new multipoint method for genome-wide association studies by imputation of genotypes (Nature Genetics)
High-resolution haplotype structure in the human genome (Nature Genetics)
Potential etiologic and functional implications of genome-wide association loci for human diseases and traits (Proceedings of the National Academy of Sciences of the United States of America)
Epidemiology and genetics of rheumatoid arthritis (Arthritis Research)
A genome-wide association study of global gene expression (Nature Genetics)
PLINK: a tool set for whole-genome association and population-based linkage analyses.
Assessing the impact of population stratification on genetic association studies (Nature Genetics)

Their papers

Partitioning heritability by functional annotation using genome-wide association summary statistics (2015) — 2,445 citations
Gene Expression Elucidates Functional Impact of Polygenic Risk for Schizophrenia (2016) — 1,080 citations
A polygenic burden of rare disruptive mutations in schizophrenia (2014) — 1,469 citations
A contribution of novel CNVs to schizophrenia from a genome-wide study of 41,321 subjects: CNV Analysis Group and the Schizophrenia Working Group of the Psychiatric Genomics Consortium (2016) — 1,284 citations
Modeling Linkage Disequilibrium Increases Accuracy of Polygenic Risk Scores (2015) — 1,263 citations
Exploring the phenotypic consequences of tissue specific gene expression variation inferred from GWAS summary statistics (2016) — 1,036 citations
Mapping genomic loci implicates genes and synaptic biology in schizophrenia (2022) — 2,226 citations
Rare coding variants in 10 genes confer substantial risk for schizophrenia (2022) — 659 citations
Genome-wide association study of over 40,000 bipolar disorder cases provides new insights into the underlying biology (2021) — 994 citations
Exome sequencing and analysis of 454,787 UK Biobank participants (2021) — 786 citations
Genetic analyses of diverse populations improves discovery for complex traits (2019) — 901 citations
Genome-wide association studies in ancestrally diverse populations: opportunities, methods, pitfalls, and recommendations. (2019) — 634 citations
Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection (2018) — 1,432 citations
Genomic dissection of bipolar disorder and schizophrenia including 28 subphenotypes (2018) — 667 citations
Systematic Evaluation of Pleiotropy Identifies 6 Further Loci Associated With Coronary Artery Disease (2017) — 244 citations
Increased burden of ultra-rare protein-altering variants among 4,877 individuals with schizophrenia (2016) — 444 citations
Partitioning heritability of regulatory and cell-type-specific variants across 11 common diseases. (2014) — 595 citations
Biological Insights From 108 Schizophrenia-Associated Genetic Loci (2014) — 7,422 citations
Five amino acids in three HLA proteins explain most of the association between MHC and seropositive rheumatoid arthritis (2012) — 954 citations
High density genetic mapping identifies new susceptibility loci for rheumatoid arthritis (2012) — 656 citations