Kate E. R. Hollinshead
Cancer Research · 2016
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Cancer Research — 34 citations into it
- Molecular Biology — 30 citations into it
Whom Kate E. R. Hollinshead trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Kate E. R. Hollinshead overall
Papers from Kate E. R. Hollinshead's perspective
Ordered by how strongly the researchers Kate E. R. Hollinshead trusts endorse each paper. Same corpus, a different order for every researcher.
- Myc Stimulates Nuclearly Encoded Mitochondrial Genes and Mitochondrial Biogenesis (Molecular and Cellular Biology)
- HIF-1-mediated expression of pyruvate dehydrogenase kinase: a metabolic switch required for cellular adaptation to hypoxia. (Cell Metabolism)
- Cancer's molecular sweet tooth and the Warburg effect. (Cancer Research)
- Why do cancers have high aerobic glycolysis? (Nature Reviews. Cancer)
- HIF-1 mediates adaptation to hypoxia by actively downregulating mitochondrial oxygen consumption. (Cell Metabolism)
- Targeting of HIF-α to the von Hippel-Lindau Ubiquitylation Complex by O2-Regulated Prolyl Hydroxylation (Science)
- Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensing. (Cell Metabolism)
- Attenuation of LDH-A expression uncovers a link between glycolysis, mitochondrial physiology, and tumor maintenance. (Cancer Cell)
- Targeting HIF-1 for cancer therapy (Nature Reviews. Cancer)
- Regulation of angiogenesis by hypoxia: role of the HIF system (Nature Medicine)
Their papers
- Hypoxia and metabolic adaptation of cancer cells (2016) — 668 citations