Matthew K. Knabel
Cancer Research · 2011
A researcher in the Science corpus: 1 paper; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Cancer Research — 15 citations into it
- Molecular Biology — 13 citations into it
Whom Matthew K. Knabel trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- G. Semenza (in Cancer Research)
- Adrian L. Harris (in Cancer Research)
Papers from Matthew K. Knabel's perspective
Ordered by how strongly the researchers Matthew K. Knabel trusts endorse each paper. Same corpus, a different order for every researcher.
- Why do cancers have high aerobic glycolysis? (Nature Reviews. Cancer)
- Transcription Factor HIF-1 Is a Necessary Mediator of the Pasteur Effect in Mammalian Cells (Molecular and Cellular Biology)
- Hypoxia — a key regulatory factor in tumour growth (Nature Reviews. Cancer)
- The M2 splice isoform of pyruvate kinase is important for cancer metabolism and tumour growth (Nature)
- HIF-1 regulates cytochrome oxidase subunits to optimize efficiency of respiration in hypoxic cells. (Cell)
- Tyrosine Phosphorylation Inhibits PKM2 to Promote the Warburg Effect and Tumor Growth (Science Signaling)
- Defining the Role of Hypoxia-Inducible Factor 1 in Cancer Biology and Therapeutics (Oncogene)
- Asparagine Hydroxylation of the HIF Transactivation Domain: A Hypoxic Switch (Science)
- HnRNP proteins controlled by c-Myc deregulate pyruvate kinase mRNA splicing in cancer (Nature)
- HIF prolyl‐hydroxylase 2 is the key oxygen sensor setting low steady‐state levels of HIF‐1α in normoxia (EMBO Journal)
Their papers
- Pyruvate Kinase M2 is a PHD3-stimulated Coactivator for Hypoxia-Inducible Factor 1 (2011) — 1,355 citations