P. Vogt
Molecular Biology · 2005–2008
A researcher in the Science corpus: 3 papers; trusts 1 researcher and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 77 citations into it; #11,523 of 17,773 by trust here, #7,124 by citations
Whom P. Vogt trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- Lewis C. Cantley (in Molecular Biology)
Who trusts P. Vogt
- J. Engelman (in Molecular Biology)
- Arkaitz Carracedo (in Molecular Biology)
- P. Pandolfi (in Molecular Biology)
Papers from P. Vogt's perspective
Ordered by how strongly the researchers P. Vogt trusts endorse each paper. Same corpus, a different order for every researcher.
- mTOR interacts with raptor to form a nutrient-sensitive complex that signals to the cell growth machinery. (Cell)
- Upstream and downstream of mTOR. (Genes & Development)
- The Mammalian Target of Rapamycin (mTOR) Partner, Raptor, Binds the mTOR Substrates p70 S6 Kinase and 4E-BP1 through Their TOR Signaling (TOS) Motif* (Journal of Biological Chemistry)
- Raptor, a binding partner of target of rapamycin (TOR), mediates TOR action. (Cell)
- A phosphatidylinositol 3-kinase/Akt pathway promotes translocation of Mdm2 from the cytoplasm to the nucleus (Proceedings of the National Academy of Sciences of the United States of America)
- Erlotinib in lung cancer - molecular and clinical predictors of outcome. (New England Journal of Medicine)
- Anticancer drugs: To the rescue? (Nature reviews. Drug discovery)
- Nuclear factor-κB inhibitors as sensitizers to anticancer drugs (Nature Reviews. Cancer)
- Enhanced sensitivity of PTEN-deficient tumors to inhibition of FRAP/mTOR (Proceedings of the National Academy of Sciences of the United States of America)
- TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (Nature Cell Biology)
Their papers
- Oncogenic PI3K deregulates transcription and translation (2005) — 817 citations
- Phosphatidylinositol 3-kinase mutations identified in human cancer are oncogenic. (2005) — 851 citations
- Class I PI3K in oncogenic cellular transformation (2008) — 616 citations