G. Peters
Molecular Biology · Nature reviews. Molecular cell biology · 2001–2006
A researcher in the Science corpus: 3 papers; trusts 1 researcher and is trusted by 9 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 31 citations into it; #4,840 of 17,773 by trust here, #7,574 by citations
- Physiology — 10 citations into it; #2,020 of 2,276 by trust here, #891 by citations
Whom G. Peters trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts G. Peters overall
Papers from G. Peters's perspective
Ordered by how strongly the researchers G. Peters trusts endorse each paper. Same corpus, a different order for every researcher.
- Bmi-1 determines the proliferative capacity of normal and leukaemic stem cells (Nature)
- Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells (Nature)
- Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation
- Oxygen sensitivity severely limits the replicative lifespan of murine fibroblasts (Nature Cell Biology)
- Loss of p16Ink4a confers susceptibility to metastatic melanoma in mice (Nature)
- Bmi1 is essential for cerebellar development and is overexpressed in human medulloblastomas (Nature)
- A DNA damage checkpoint response in telomere-initiated senescence (Nature)
- Cellular senescence as a tumor-suppressor mechanism. (Trends in Cell Biology)
- Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a). (Molecules and Cells)
- Reactive oxygen species act through p38 MAPK to limit the lifespan of hematopoietic stem cells (Nature Medicine)
Their papers
- Regulation of the INK4b–ARF–INK4a tumour suppressor locus: all for one or one for all (2006) — 891 citations
- Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence (2001) — 694 citations
- Glycolytic enzymes can modulate cellular life span. (2005) — 661 citations