P. Velasco

Physiology · 2004–2009

A researcher in the Science corpus: 4 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

• Physiology — 51 citations into it
• Cellular and Molecular Neuroscience — 39 citations into it

Whom P. Velasco trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. D. Selkoe
2. W. Klein

Who trusts P. Velasco overall

1. F. D. De Felice

Papers from P. Velasco's perspective

Ordered by how strongly the researchers P. Velasco trusts endorse each paper. Same corpus, a different order for every researcher.

1. Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
2. Pathways towards and away from Alzheimer's disease (Nature)
3. Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction. (Neuron)
4. Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo (Nature)
5. Immunization reverses memory deficits without reducing brain Aβ burden in Alzheimer's disease model (Nature Neuroscience)
6. A specific amyloid-beta protein assembly in the brain impairs memory. (Nature)
7. Morphological changes in dendritic spines associated with long-term synaptic plasticity. (Annual Review of Neuroscience)
8. Dendritic spine pathology: cause or consequence of neurological disorders? (Brain research. Brain research reviews)
9. Protofibrils, pores, fibrils, and neurodegeneration: separating the responsible protein aggregates from the innocent bystanders. (Annual Review of Neuroscience)
10. Altered expression of synaptic proteins occurs early during progression of Alzheimer’s disease (Neurology)

Their papers

• Protection of synapses against Alzheimer's-linked toxins: Insulin signaling prevents the pathogenic binding of Aβ oligomers (2009) — 616 citations
• Aβ Oligomers Induce Neuronal Oxidative Stress through an N-Methyl-D-aspartate Receptor-dependent Mechanism That Is Blocked by the Alzheimer Drug Memantine* (2007) — 904 citations
• Aβ Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease (2007) — 1,061 citations
• Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers (2004) — 861 citations