William Pao
Pulmonary and Respiratory Medicine · 2004–2014
A researcher in the Science corpus: 27 papers; trusts 10 researchers and is trusted by 14 researchers here, as inferred from the citation graph.
Subfields
- Pulmonary and Respiratory Medicine — 285 citations into it; #45 of 770 by trust here, #5 by citations
- Oncology — 266 citations into it; #422 of 1,698 by trust here, #69 by citations
- Molecular Biology — 223 citations into it
- Hematology — 27 citations into it
Whom William Pao trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Vincent A. Miller
- I. Weinstein
- L. Tanoue
- J. Mendelsohn
- M. Kris
- Gregory Riely
- J. Engelman
- C. Sawyers
- D. Bell
- C. Yun
Who trusts William Pao overall
- B. Chan
- Carlos L. Arteaga
- J. Engelman
- Vincent A. Miller
- Gregory Riely
- Adi F. Gazdar
- C. Sawyers
- J. Baselga
- H. Shigematsu
- J. Markman
Papers from William Pao's perspective
Ordered by how strongly the researchers William Pao trusts endorse each paper. Same corpus, a different order for every researcher.
- Clinical Resistance to STI-571 Cancer Therapy Caused by BCR-ABL Gene Mutation or Amplification (Science)
- EGF receptor gene mutations are common in lung cancers from "never smokers" and are associated with sensitivity of tumors to gefitinib and erlotinib. (Proceedings of the National Academy of Sciences of the United States of America)
- Targeted cancer therapy (Nature)
- Gefitinib-Sensitizing EGFR Mutations in Lung Cancer Activate Anti-Apoptotic Pathways (Science)
- Mutations of the BRAF gene in human cancer (Nature)
- Activating mutations in the epidermal growth factor receptor underlying responsiveness of non-small-cell lung cancer to gefitinib. (New England Journal of Medicine)
- Hepatocyte growth factor induces gefitinib resistance of lung adenocarcinoma with epidermal growth factor receptor-activating mutations. (Cancer Research)
- Novel D761Y and Common Secondary T790M Mutations in Epidermal Growth Factor Receptor–Mutant Lung Adenocarcinomas with Acquired Resistance to Kinase Inhibitors (Clinical Cancer Research)
- Structure of the Epidermal Growth Factor Receptor Kinase Domain Alone and in Complex with a 4-Anilinoquinazoline Inhibitor* (Journal of Biological Chemistry)
- Clinical Course of Patients with Non–Small Cell Lung Cancer and Epidermal Growth Factor Receptor Exon 19 and Exon 21 Mutations Treated with Gefitinib or Erlotinib (Clinical Cancer Research)
Their papers
- Somatic mutations affect key pathways in lung adenocarcinoma (2008) — 2,751 citations
- Characterizing the cancer genome in lung adenocarcinoma (2007) — 1,146 citations
- Mapping the hallmarks of lung adenocarcinoma with massively parallel sequencing (2012) — 1,811 citations
- KRAS Mutations and Primary Resistance of Lung Adenocarcinomas to Gefitinib or Erlotinib (2005) — 1,447 citations
- Anchored multiplex PCR for targeted next-generation sequencing (2014) — 832 citations
- Using Multiplexed Assays of Oncogenic Drivers in Lung Cancers to Select Targeted Drugs (2014) — 1,641 citations
- Frequent and Focal FGFR1 Amplification Associates With Therapeutically Tractable FGFR1 Dependency in Squamous-cell Lung Cancer (2010) — 861 citations
- New driver mutations in non-small-cell lung cancer. (2011) — 1,202 citations
- Acquired resistance to TKIs in solid tumours: learning from lung cancer (2014) — 819 citations
- AZD9291, an irreversible EGFR TKI, overcomes T790M-mediated resistance to EGFR inhibitors in lung cancer (2014) — 1,855 citations
- Analysis of Tumor Specimens at the Time of Acquired Resistance to EGFR TKI therapy in 155 patients with EGFR mutant Lung Cancers (2013) — 2,352 citations
- Integrative genome analyses identify key somatic driver mutations of small cell lung cancer (2012) — 1,318 citations
- Comprehensive genomic characterization of squamous cell lung cancers (2012) — 3,784 citations
- ROS1 rearrangements define a unique molecular class of lung cancers. (2012) — 728 citations
- HER2 amplification: a potential mechanism of acquired resistance to EGFR inhibition in EGFR-mutant lung cancers that lack the second-site EGFRT790M mutation. (2012) — 666 citations
- Clinical definition of acquired resistance to epidermal growth factor receptor tyrosine kinase inhibitors in non-small-cell lung cancer. (2010) — 798 citations
- Acquired resistance to EGFR tyrosine kinase inhibitors in EGFR mutant lung cancer: Distinct natural history of patients with tumors harboring the T790M mutation (2010) — 619 citations
- Rational, biologically based treatment of EGFR-mutant non-small-cell lung cancer (2010) — 1,084 citations
- American Society of Clinical Oncology Clinical Practice Guideline update on chemotherapy for stage IV non-small-cell lung cancer. (2009) — 936 citations
- MET amplification occurs with or without T790M mutations in EGFR mutant lung tumors with acquired resistance to gefitinib or erlotinib (2007) — 1,698 citations