S. Venneti
Molecular Biology · 2017
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 38 citations into it
- Cancer Research — 35 citations into it
Whom S. Venneti trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- Craig B. Thompson (in Cancer Research)
Papers from S. Venneti's perspective
Ordered by how strongly the researchers S. Venneti trusts endorse each paper. Same corpus, a different order for every researcher.
- Beyond aerobic glycolysis: Transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis (Proceedings of the National Academy of Sciences of the United States of America)
- Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity (Proceedings of the National Academy of Sciences of the United States of America)
- Myc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addiction (Proceedings of the National Academy of Sciences of the United States of America)
- The common feature of leukemia-associated IDH1 and IDH2 mutations is a neomorphic enzymatic activity that converts α-ketoglutarate to 2-hydroxyglutarate (Cancer Cell)
- Cancer-associated IDH1 mutations produce 2-hydroxyglutarate (Nature)
- Hallmarks of cancer: the next generation. (Cell)
- IDH1 and IDH2 mutations in gliomas. (New England Journal of Medicine)
- Reductive carboxylation supports growth in tumor cells with defective mitochondria (Nature)
- Glutamine supports pancreatic cancer growth through a Kras-regulated metabolic pathway (Nature)
- An Integrated Genomic Analysis of Human Glioblastoma Multiforme (Science)
Their papers
- Glutaminolysis: A Hallmark of Cancer Metabolism. (2017) — 763 citations