P. Soares
Molecular Biology · Nature Communications · 2003–2013
A researcher in the Science corpus: 3 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 63 citations into it
- Epidemiology — 5 citations into it
Whom P. Soares trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts P. Soares overall
Papers from P. Soares's perspective
Ordered by how strongly the researchers P. Soares trusts endorse each paper. Same corpus, a different order for every researcher.
- TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
- Phosphorylation and functional inactivation of TSC2 by Erk implications for tuberous sclerosis and cancer pathogenesis. (Cell)
- Phosphorylation and Regulation of Akt/PKB by the Rictor-mTOR Complex (Science)
- Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control. (Molecules and Cells)
- TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (Nature Cell Biology)
- TOR signaling in growth and metabolism. (Cell)
- The TSC1-2 tumor suppressor controls insulin–PI3K signaling via regulation of IRS proteins (Journal of Cell Biology)
- Rictor, a novel binding partner of mTOR, defines a rapamycin-insensitive and raptor-independent pathway that regulates the cytoskeleton. (Current Biology)
- Mammalian cell size is controlled by mTOR and its downstream targets S6K1 and 4EBP1/eIF4E. (Genes & Development)
- Rheb GTPase is a direct target of TSC2 GAP activity and regulates mTOR signaling. (Genes & Development)
Their papers
- The mTOR Signalling Pathway in Human Cancer (2012) — 795 citations
- Frequency of TERT promoter mutations in human cancers (2013) — 850 citations
- BRAF mutations and RET/PTC rearrangements are alternative events in the etiopathogenesis of PTC (2003) — 696 citations