Ischemic tolerance and endogenous neuroprotection.
By U. Dirnagl, Roger Simon, J. Hallenbeck. Trends in Neurosciences, 2003
837 citations recorded by the source corpus.
Subfields: Developmental Neuroscience, Molecular Biology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/fd1c6427f8791097ab30426541fd83ddb8bbf59f
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Citations in the corpus
- Glucocorticoids, depression, and mood disorders: structural remodeling in the brain. (2005, background): “Yet, the brain protects itself, as for example, in the phenomenon of ischemia preconditioning [25,26]; namely, that prior stimulation of the hippocampus by a small ischemic event can induce a protective mechanism that may reduce the damage produced by a full-scale ischemic event.”
- A new penumbra: transitioning from injury into repair after stroke (2008, background): “? Even during the early stages of cerebral ischemia, a complex network of endogenous neuroprotective responses may be triggere”
- Cerebral preconditioning and ischaemic tolerance (2006, neutral): “Preconditioning stimuli Differences in the intensity, duration, and/or frequency of a particular stress stimulus determine whether that stimulus is too weak to elicit any response, of sufficient magnitude to serve as a preconditioning trigger, or too robust and therefore harmful (for a review, se”
- Astrocyte reactivity and reactive astrogliosis: costs and benefits. (2014, background): “This phenomenon is known as ischemic preconditioning or ischemic tolerance and offers an opportunity for designing novel treatment strategies (59).”
- Renal ischemia/reperfusion injury; from pathophysiology to treatment (2015, background): “Ischemic preconditioning Ischemic preconditioning (IP) is a tolerance or adaptability of organ or tissue after primary exposure to a brief ischemia stimulus (124).”
- Cellular and molecular mechanisms of neural repair after stroke: Making waves (2006, background): “EPO is induced near the infarct after stroke(73,74) (see Fig 2) and is under the control of the hypoxia-inducible factor 1 transcription factor, an early molecular signal after stroke.(73,74) In vitro evidence suggests that EPO promotes neuronal differentiation of neural stem cells.”
- Neuroprotection for ischemic stroke: past, present and future. (2008, neutral): “…Gene expression (MacManus et al., 1997; Sharp et al., 1999; Sharp et al., 2000; Read et al., 2001) Ischemic tolerance; preconditioning (Kirino, 2002; Dirnagl et al., 2003; Gidday, 2006; Stenzel-Poore et al., 2007; PerezPinzon, 2007) Mitochondria (Fiskum, 2000; Friberg et al., 2002) Cerebral…”