Cellular and molecular mechanisms of neural repair after stroke: Making waves

By S. Carmichael. Annals of Neurology, 2006

595 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Developmental Neuroscience

View Paper: https://www.semanticscholar.org/paper/330a4aa81d9ae93d0cbf24cd7ec5e8e90412099a

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In Cellular and Molecular Neuroscience

In Developmental Neuroscience

Citations in the corpus

Astrocytes: biology and pathology (2009, background): “Rather, the concept of the ‘neurovascular unit’ takes an integrated view of a given brain parenchymal blood vessel and the glial elements that surround it [38, 139, 206].”
Activity-dependent structural plasticity. (2009, background): “Post-lesion disinhibition goes along with alterations at cellular and molecular levels (Carmichael, 2006) as well as with reactive sprouting and synaptogenesis (Stroemer et al., 1995).”
Plasticity during stroke recovery: from synapse to behaviour (2009, background): “Although we have described some of the molecules implicated in the recovery process, we direct readers to other recent articles that focus more exclusively on this subjec”
Repairing the human brain after stroke: I. Mechanisms of spontaneous recovery (2008, background): “In animals, this periinfarct zone often shows the greatest levels of growth-related molecular changes after stroke.(25,29,38,39,46,89) –99 Furthermore, amplification of periinfarct repair-related events in animals by a therapeutic intervention has been associated with improved behavioral outcome.”
Reactive gliosis and the multicellular response to CNS damage and disease (2014, unknown): “for formation of new relay circuits (Bareyre et al., 2004; Carmichael, 2006; Clarkson et al., 2010; Courtine et al., 2008; Li et al., 2010; Overman et al., 2012).”
Agreed definitions and a shared vision for new standards in stroke recovery research: The Stroke Recovery and Rehabilitation Roundtable taskforce (2017, background): “In animals, this term refers to improvements in recovery of behavior in the absence of a specific, targeted treatment and occurs during a time-sensitive window that begins early after stroke and slowly tapers off.(6,11,14) In human stroke survivors, a similar period of heightened recovery of behavio”
Postnatal deletion of Numb/Numblike reveals repair and remodeling capacity in the subventricular neurogenic niche. (2006, background): “It has been reported that migration of SVZ neuroblasts to distant sites can be caused by brain injury such as stroke (reviewed in Lindvall and Kokaia, 2004; Carmichael, 2006).”