Virulence factors of Candida albicans.
By R. Calderone, W. Fonzi. Trends in Microbiology, 2001
1,391 citations recorded by the source corpus.
Subfields: Epidemiology, Infectious Diseases
View Paper: https://www.semanticscholar.org/paper/fa1dee9764434408294cc6a055790080b0998921
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Epidemiology
In Infectious Diseases
Citations in the corpus
- Dectin‐1 mediates macrophage recognition of Candida albicans yeast but not filaments (2005, background): “…to rapidly and reversibly switch between yeast and filamentous morphologies is cru- cial to pathogenicity, and it is thought that the filamentous morphology provides some advantage during interaction with the mammalian immune system (Lo et al, 1997; Calderone and Fonzi, 2001; Saville et al, 2003).”
- Candida albicans Secreted Aspartyl Proteinases in Virulence and Pathogenesis (2003, background): “albicans, hyphal formation, surface recognition molecules, phenotypic switching, and extracellular hydrolytic enzyme production have been the most widely studied in recent years (24).”
- Candida species: current epidemiology, pathogenicity, biofilm formation, natural antifungal products and new therapeutic options. (2013, background): “Candida species are considered important pathogens due to their versatility and ability to survive in various anatomical sites (Calderone & Fonzi, 2001).”
- The Selective Value of Bacterial Shape (2006, background): “Although shape changes are clearly associated with virulence, the reduced pathogenicity of any particular mutant cannot yet be attributed to shape alone (231, 276), leading some to conclude that there is still “no unequivocal demonstration” (37) of an explicit cause-and-effect relationship between f”
- Neutrophil Extracellular Traps Contain Calprotectin, a Cytosolic Protein Complex Involved in Host Defense against Candida albicans (2009, background): “The ability to reversibly switch between different morphologies upon external stimuli appears to be essential for the virulence of C. albicans [22,23].”
- A Pseudomonas aeruginosa quorum‐sensing molecule influences Candida albicans morphology (2004, background): “Third, a number of C. albicans eukaryotic cell adhesins (Staab et al ., 1999; Fu et al ., 2002) and virulence-related gene products (van Burik and Magee, 2001; Calderone and Fonzi, 2001) are more abundant in the filamentous form of the fungus.”