The tissue distribution of the mRNA of ghrelin and subtypes of its receptor, GHS-R, in humans.
By S. Gnanapavan, B. Kola, Stephen A. Bustin, D. Morris, Patrick J. McGee, P. Fairclough, S. Bhattacharya, R. Carpenter, A. Grossman, M. Korbonits. Journal of Clinical Endocrinology and Metabolism, 2002
1,486 citations recorded by the source corpus.
Subfields: Endocrine and Autonomic Systems, Endocrinology, Diabetes and Metabolism, Physiology
View Paper: https://www.semanticscholar.org/paper/f93324f07ce284719ed94f9f3b23a2815fd69b73
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Citations in the corpus
- Ghrelin and des-acyl ghrelin inhibit cell death in cardiomyocytes and endothelial cells through ERK1/2 and PI 3-kinase/AKT (2002, background): “…cardiomyocytes provides further support to the hypothesis that multiple ghrelin recep- 1036 The Journal of Cell Biology | Volume 159, Number 6, 2002 tors may be expressed in the cardiovascular system (Papotti et al., 2000; Bodart et al., 2002; Gnanapavan et al., 2002; Katugampola et al., 2001).”
- Ghrelin: structure and function. (2005, background): “Ghrelin mRNA is expressed in the kidney, especially in the glomeruli (89, 162).”
- Deletion of Ghrelin Impairs neither Growth nor Appetite (2003, methodology): “GHS-R is predominantly expressed in the pituitary and brain (9, 11), but ghrelin is expressed in almost all tissues, with the highest expression in the stomach (9, 15).”
- Nutritional controls of beef cow reproduction (2005, background): “, 2001), lower amounts are derived from the intestines, kidney, immune system, placenta, pituitary, gonads, and hypothalamus (Gnanapavan et al., 2002).”
- Expression of ghrelin receptor mRNA in the rat and the mouse brain (2006, background): “…Western blot analysis, and in situ hybridization histochemistry (ISHH) (Howard et al., 1996; Guan et al., 1997; Bennett et al., 1997; Tannenbaum et al., 1998; Willesen et al., 1999; Kamegai et al., 1999; Katayama et al., 2000; Mitchell et al., 2001; Shuto et al., 2001; Gnanapavan et al., 2002).”
- Ghrelin inhibits leptin- and activation-induced proinflammatory cytokine expression by human monocytes and T cells. (2004, methodology): “Primers for human IL-1β, IL-6, and TNF-α genes and GAPDH as control were purchased from Biosource International (Camarillo, California, USA); human GHS-R 1a and ghrelin were used as described previously (9).”
- Biological, physiological, pathophysiological, and pharmacological aspects of ghrelin. (2004, background): “(22, 30, 128, 169, 206), ghrelin has been demonstrated to be expressed by pancreatic endocrine -cells in rat and human tissue by some authors (206) and by pancreatic -cells by one group (169).”