Fundamentals of endothelial function for the clinical cardiologist.

By S. Verma, T. Anderson. Circulation, 2002

667 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Physiology

View Paper: https://www.semanticscholar.org/paper/f8a860c1cf98f009ca12b624af66139d2dce7496

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. S. Verma
2. T. Anderson
3. E. Schiffrin

In Physiology

1. K. Sugamura
2. E. Schiffrin
3. J. Keaney

Citations in the corpus

• Endothelial function testing as a biomarker of vascular disease. (2003, background): “The rationale for why endothelial function testing, either directly or indirectly, may serve as an indicator of vascular damage or disease is as follows: (1) The healthy endothelium is nonthrombogenic; (2) endothelial dysfunction occurs in response to vascular risk factors and is an early event in a”
• Oxidative stress and reactive oxygen species in endothelial dysfunction associated with cardiovascular and metabolic diseases. (2018, background): “vasodilator and vasoconstrictor factors, which predisposes the vasculature towards a pro-thrombotic and pro-atherogenic phenotype, characterized by vasoconstrinction, leukocyte adhesion, platelet activation, mitogenesis, pro-oxidation, impaired coagulation, vascular inflammation, atherosclerosis and”
• Reactive Oxygen Species in Cardiovascular Disease (2011, background): “Also among the features of reperfusion injury is a burst of free radical formation that may continue for hours [230] and has been attributed to multiple sources including xanthine oxidase, activated neutrophils, electron leakage from mitochondria, catecholamine oxidation, cyclooxygenase and lipoxyge”
• Pathophysiology of Atherosclerosis (2022, background): “Briefly, when ECs lose their ability to maintain homeostasis, vessel walls are predisposed to vasoconstriction, lipid infiltration, leukocyte adhesion, platelet activation, and oxidative stress, among other things [23,24].”
• Endothelial dysfunction. (2004, background): “Vasoconstrictors include endothelin-1 (ET-1), angiotensin II (Ang II), thromboxane A2, and reactive oxygen species (ROS) (2,3).”