Time to give up on a single explanation for autism

By F. Happé, A. Ronald, Robert Plomin. Nature Neuroscience, 2006

1,022 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Education, Genetics

View Paper: https://www.semanticscholar.org/paper/f5de40a8b498e99304f994a69c4d2dac46560c50

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Citations in the corpus

Embodiment and sense-making in autism (2013, background): “Given their cognitivist and functionalist background, it is no surprise that the accounts consider perception, action, and cognition as relatively separate domains that can be investigated practically in isolation (Frith, 2003; Happé et al., 2006).”
Autistics speak but are they heard (2013, neutral): “There exists no definitive account of its development, and current research points away from a single ‘cause’ towards a range of potential neurodevelopmental differences (Happe et al., 2006) - to say nothing of variations in social and cultural circumstances in which autistic people live.”
Vision in autism spectrum disorders. (2009, background): “We have seen that diagnostic measures (Asperger vs. autism), or performance measures (Block Design peaks, Famous face recognition, Verbal IQ) can be useful here (see also Happé et al., 2006), but obviously this technique increases the problem of participant recruitment.”
Autism spectrum disorders: developmental disconnection syndromes (2007, background): “com repetitive behaviors [60], rather than to the traditional clinical diagnosis of the autisms [6].”
Cognitive theories of autism (2007, background): “To investigate this possibility, Booth, Charlton, Hughes, and Happé (2003) compared performance on a drawing task, which required the EF element of planning and central coherence, between boys with an autistic disorder, boys with ADHD and boys who were developing typically.”
Agenesis of the corpus callosum: genetic, developmental and functional aspects of connectivity (2007, neutral): “Psychiatric diagnoses are based on clusters of behaviors, which are very complex and likely involve multiple neural mechanisms 108 .”