ERRATA

By Y. Kureishi, Zhengyu Luo, Anna Bialik, D. Fulton, D. Lefer, W. Sessa, François Romagne, Marc Bonneville. Nature Medicine, 2001

1,486 citations recorded by the source corpus.

Subfields: Cancer Research, Molecular Biology, Surgery

View Paper: https://www.semanticscholar.org/paper/f59a291d6515eeef0e39e007b54aa24a1fcedd45

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cancer Research

1. J. Liao
2. W. Sessa

In Molecular Biology

1. D. Yellon
2. D. Hausenloy
3. J. Liao
4. U. Förstermann
5. W. Sessa
6. E. Murphy
7. C. Steenbergen
8. M. Mocanu

In Surgery

1. J. Liao
2. U. Förstermann
3. W. Sessa

Citations in the corpus

• Endothelial dysfunction - a major mediator of diabetic vascular disease. (2013, background): “Statins can also directly activate eNOS via post-translational mechanisms involving activation of the phosphatidylinositol 3-kinase/protein kinase Akt pathway [176].”
• Nitric oxide synthases: regulation and function. (2012, background): “Statins increase the expression of eNOS,(148) but also enhance eNOS activity by decreasing caveolin abundance(149) and by activation of the phosphatidylinositol 3-kinase/Akt pathway.(150) Several statins inhibit endothelial O2 2† formation by reducing the expression and/or activity of NADPH oxidase ”
• Pleiotropic effects of statin therapy: molecular mechanisms and clinical results. (2008, background): “Third, statins can activate the phosphatidylinositol 3-kinase (PI3K)/protein kinase Akt pathway [77].”
• New directions for protecting the heart against ischaemia-reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway. (2004, background): “HMG-CoA reductase inhibitors have also been shown to up-regulate the PI3K–Akt kinase cascade in endothelial cell lines [99].”
• Therapeutic Strategies for Enhancing Angiogenesis in Wound Healing (2018, background): “Used with permission from [2 studies suggest that statins angiogenic activity is mediated via the Akt/ PI3K pathway to regulate proliferation of endothelial cells and capillary morphogenesis, and to modulate secretion of critical factors such as VEGF-A in multiple cell types [261].”