Recent insights into hepatic lipid metabolism in non-alcoholic fatty liver disease (NAFLD).

By G. Musso, R. Gambino, M. Cassader. Progress in lipid research, 2009

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Interactions between gut microbiota and host metabolism predisposing to obesity and diabetes. (2011, background): “This eventually resulted in accumulation of hepatic and adipose tissue triglycerides (56, 59) (Table 1).”
Hepatic lipotoxicity and the pathogenesis of nonalcoholic steatohepatitis: The central role of nontriglyceride fatty acid metabolites (2010, background): “1).(62) Adipose tissue lipolysis is the major source of fatty acids delivered to the liver, whereas DNL normally contributes only about 5%; in patients with NASH, DNL may contribute up to 26% of the fatty acids.”
Triglyceride metabolism in the liver (2017, neutral): “Additional pathways for FA oxidation include α -oxidation and ω -oxidation within the ER and are mediated by cytochrome P450 4A family members (153, 187) (Fig.”