Nitric oxide in the pathogenesis of vascular disease

By Huige Li, U. Förstermann. Journal of Pathology, 2000

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Nitric oxide synthases: regulation and function. (2012, background): “Based on the combination of those effects, NO produced in endothelial cells can be considered an anti-atherosclerotic principle (for review, see Li and Fo¨rstermann 162 ). intracellular Ca 2 + concentrations (below 40 nM) due to a different amino acid structure of the calmodulin-binding site.”
Atherosclerosis: Process, Indicators, Risk Factors and New Hopes (2014, background): “C‐reactive protein induces platelets binding to endothelial cells([78]) and stimulates monocytes and lymphocytes absorption into endothelial walls.”
Vascular oxidative stress, nitric oxide and atherosclerosis. (2014, background): “Under physiological conditions, eNOS produces NO, w hich represents a key element in the vasoprotective function of the endothelium [60-62].”
Hyperglycemia inhibits endothelial nitric oxide synthase activity by posttranslational modification at the Akt site. (2001, background): “Finally, endothelial cell nitric oxide inhibits proliferation of vascular smooth muscle cells (6).”
Free radicals and antioxidants in normal physiological functions and human disease. (2007, background): “…of O2•− and NO• (leading to peroxynitrite formation) seems to be also involved in of Bioc t a e f e f t t t i i fi A fl t i i l h e b e f ( h s p e s i t l C c ( R f d t m t i w t b n o 2 i f M. Valko et al. / The International Journal he process of hypertension (Li & Forstermann, 2000).”