Stress and the inflammatory response: a review of neurogenic inflammation.

By P. Black. Brain, behavior, and immunity, 2002

668 citations recorded by the source corpus.

Subfields: Behavioral Neuroscience, Biological Psychiatry, Cellular and Molecular Neuroscience

View Paper: https://www.semanticscholar.org/paper/f08f126312d2176607ca499d6537e7b4893b535f

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Behavioral Neuroscience

1. Melissa Bateson
2. Colin M. Beale
3. N. Woo
4. E. Deane
5. J. Pickup

In Biological Psychiatry

1. N. Woo
2. E. Deane
3. J. Pickup

In Cellular and Molecular Neuroscience

1. N. Woo
2. E. Deane
3. J. Pickup

Citations in the corpus

• Burnout and risk of cardiovascular disease: evidence, possible causal paths, and promising research directions. (2006, background): “Malden, MA: Blackwell. Smith, T. W., & Ruiz, J. M. (2002). Psychosocial influences on the development and course of coronary heart disease: Current status and implications for research and practice.”
• Anthropogenic Noise as a Stressor in Animals: A Multidisciplinary Perspective (2007, background): “For example, long-term activation of the fight-or-flight response across the life span can lead to coronary dysfunction and disease (see Romero & Butler, this issue), an effect that may involve vascular inflammation as an intermediate mechanism (Black, 2002, 2003).”
• Inflammation and activated innate immunity in the pathogenesis of type 2 diabetes. (2004, background): “Elevated portal bloodstream LPS levels stimulate Kupffer cell receptors and cytokine release (27).”
• Depression, anxiety, and stress and the distinction between intentional and unintentional mind wandering. (2019, background): “This damage is further exacerbated by stress-related increases in inflammation (Black, 2002; Raison, Capuron, & Miller, 2006).”
• Interactions between Temperament, Stress, and Immune Function in Cattle (2011, background): “Stressors are any internal or external stimuli or threat (physical, psychological, or chemical) that disrupts homeostasis [22, 23].”
• Modulation of fish growth hormone levels by salinity, temperature, pollutants and aquaculture related stress: a review (2009, background): “Stress is well known to affect GH synthesis, release and circulating levels in mammals (De Feo 1996; Black 2002; Stokes 2003), but a similar consideration of GH as a stress-related hormone, in fish, is not well recognized.”