Junction adhesion molecule is a receptor for reovirus.
By E. Barton, J. Forrest, J. Connolly, J. Chappell, Yuan Liu, F. Schnell, A. Nusrat, C. Parkos, T. Dermody. Cell, 2001
651 citations recorded by the source corpus.
Subfields: Epidemiology, Immunology, Infectious Diseases
View Paper: https://www.semanticscholar.org/paper/ef3aa9283c7db4c89c28cae67826ba87d3a9c1e2
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In Immunology
Citations in the corpus
- β1 Integrin Mediates Internalization of Mammalian Reovirus (2006, background): “In support of a role for coreceptor utilization in reovirus growth, reovirus infection can occur in the absence of 1 (19) or JAM-A (5), albeit at greatly reduced efficiency.”
- Riding the DUBway: regulation of protein trafficking by deubiquitylating enzymes (2006, positive): “Refl ecting these varied biological roles, the Nox proteins have been implicated in several cell-fate pathways, such as the Ras mitogenic pathway (Irani et al., 1997), the MAP kinases (Gu et al., 2002; Xu et al., 2002), the JAK–STAT pathways (Schieffer et al., 2000), and NF-κB (Sulciner et al.,…”
- Proinflammatory Cytokines Disrupt Epithelial Barrier Function by Apoptosis-Independent Mechanisms 1 (2003, background): “The functional significance of altered JAM-1 expression in membrane rafts may relate to its status as a putative target for pathogens such as reovirus (62).”
- The tight junction: a multifunctional complex. (2004, background): “This interaction stimulates nuclear factor- B (NF- B) activation, which results in cellular apoptosis (16).”
- Endothelial cell-to-cell junctions: molecular organization and role in vascular homeostasis. (2004, background): “Interestingly, JAM-A may also mediate heterophilic adhesion to ligands as diverse as the leukocyte L 2-integrin (249) and the attachment protein sigma1 of Reovirus (28).”
- Reovirus Outer Capsid Protein μ1 Induces Apoptosis and Associates with Lipid Droplets, Endoplasmic Reticulum, and Mitochondria (2006, background): “tosis induction in cultured cells (4).”