Endocrine regulation of male fertility by the skeleton.

By Franck Oury, G. Sumara, O. Sumara, Mathieu Ferron, Haixin Chang, Charles E. Smith, L. Hermo, S. Suarez, Bryan L. Roth, P. Ducy and 1 more. Cell, 2011

613 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Orthopedics and Sports Medicine, Reproductive Medicine

View Paper: https://www.semanticscholar.org/paper/7ec301e4fa8f4f4738ebd12dda5ab467e104cf35

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Citations in the corpus

Biology of Bone Tissue: Structure, Function, and Factors That Influence Bone Cells (2015, background): “In the testis, osteocalcin can bind to a specific receptor in Leydig cells and enhances testosterone synthesis and, consequently, increases fertility [206].”
Molecular Mechanisms of Vitamin D Action (2012, background): “Finally, osteocalcin has been identified by Karsenty and coworkers [36] as a bonesecreted hormone that improves insulin release from pancreatic b-cells, increases insulin metabolic responsiveness, and is required for optimal fertility in male mice [37].”
Regulation of the Hippo-YAP pathway by G-protein coupled receptor signaling (2012, background): “Indeed, it has been shown that knockout of gprc6a in Leydig cells reduces testis size (Oury et al., 2011).”