Monocyte and macrophage plasticity in tissue repair and regeneration.
By Amitava Das, M. Sinha, Soma Datta, Motaz Abas, Scott Chaffee, Chandan K. Sen, Sashwati Roy. American Journal of Pathology, 2015
676 citations recorded by the source corpus.
Subfields: Immunology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/a33e53b447d54804bef529f49e9c8469516dd7e3
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In Immunology
In Molecular Biology
Citations in the corpus
- Macrophage plasticity, polarization, and function in health and disease (2018, background): “During resolution, M2 macrophages are a major source of lipid mediators and produce anti-inflammatory cytokines, IL-10 and TGF-β involved in tissue resolution (Das et al., 2015; Ginhoux & Jung, 2014; Hinz et al., 2012; Mantovani et al., 2013).”
- Skin Acute Wound Healing: A Comprehensive Review (2019, background): “M2b and M2c macrophages mostly contribute to resolving the inflammatory phase by reducing the damage caused by prolonged activation of M1 macrophages [67, 83, 84], and driving the resolution of inflammation.”
- Macrophage Phenotypes Regulate Scar Formation and Chronic Wound Healing (2017, background): “Different wound macrophage subsets could be derived from monocytes with different phenotypes recruited at different times that differentiate into macrophages with distinct phenotypes, or monocytes could be recruited at different times in the course of wound healing where the ever-changing wound mili”
- Macrophages, Foreign Body Giant Cells and Their Response to Implantable Biomaterials (2015, background): “However, the cells are more associated with repair than defense [38,39].”
- Platelet-Rich Plasma: New Performance Understandings and Therapeutic Considerations in 2020 (2020, background): “A typical feature of the MPS is the plasticity in their gene expression patterns and functional overlap between these cell types.”
- Wound healing: cellular mechanisms and pathological outcomes (2020, background): “It may additionally be driven by miRNAs [31], transcription factors [41], and modulation of pro-inflammatory and anti-inflammatory receptors [41,42].”