Molecular physiology of low-voltage-activated t-type calcium channels.

By E. Perez-Reyes. Physiological Reviews, 2003

1,685 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Cellular and Molecular Neuroscience, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/e8675ceaf5137b3d761210406b5d157edcef8998

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. J. Nerbonne
2. R. Kass
3. Rui Wang
4. Brett A. Simms
5. G. Zamponi
6. William A. Catterall
7. E. Perez-Reyes
8. F. H. Yu

In Cellular and Molecular Neuroscience

1. William A. Catterall
2. R. Kass
3. J. Nerbonne
4. F. H. Yu
5. Rui Wang
6. G. Zamponi
7. T. Scheuer
8. Brett A. Simms

In Molecular Biology

1. William A. Catterall
2. J. Nerbonne
3. R. Kass
4. F. H. Yu
5. Rui Wang
6. G. Zamponi
7. T. Scheuer
8. Brett A. Simms

Citations in the corpus

• International Union of Pharmacology. XLVIII. Nomenclature and Structure-Function Relationships of Voltage-Gated Calcium Channels (2005, background): “Platzer J, Engel J, Schrott-Fischer A, Stephan K, Bova S, Chen H, Zheng H, and Striessnig J (2000) Congenital deafness and sinoatrial node dysfunction in mice lacking class D L-type calcium channels.”
• Physiological implications of hydrogen sulfide: a whiff exploration that blossomed. (2012, background): “T-type VDCC also have critical roles to play in the processing of either somatic (479) or visceral (305) nociceptive information and in control of pain (301).”
• The neurobiology of antiepileptic drugs (2004, neutral): “In contrast to HVA calcium channels, whose main function is regulation of neurotransmitter release, low voltage-activated (T-type) calcium channels are believed to regulate neuronal firing by participating in bursting and intrinsic oscillation”
• Molecular physiology of cardiac repolarization. (2005, methodology): “LVA Cav channels, also referred to as T-type Ca(2) channels (394), activate at relatively hyperpolarized membrane potentials, i.”
• Computational Methods in Drug Discovery (2014, background): “T-type calcium channels underlie the generation of rhythmical firing patters in the central nervous system and have been implicated in the pathologies of epilepsy and neuropathic pain (Huguenard and Prince, 1992; Perez-Reyes, 2003; Bourinet and Zamponi, 2005).”
• Neuronal voltage-gated calcium channels: structure, function, and dysfunction. (2014, background): “Cav3 channels can be distinguished by their sensitivity to nickel and relative resistance to block by cadmium ions, which block all HVA channels in the low micromolar range (for review, see Perez-Reyes, 2003).”
• Voltage-gated ion channels and gating modifier toxins. (2007, background): “3) conduct T-type calcium currents that are important for repetitive action potential firing of neurons in the brain and in the pacemaker cells of the sino-atrial node in the heart (Perez-Reyes, 2003).”
• Regulation of Insulin Synthesis and Secretion and Pancreatic Beta-Cell Dysfunction in Diabetes (2012, neutral): “The LVA open transiently upon modest depolarization [176, 177].”