Oxidative stress and nuclear factor-kappaB activation: a reassessment of the evidence in the light of recent discoveries.
By A. Bowie, Luke A. O'Neill. Biochemical Pharmacology, 2000
904 citations recorded by the source corpus.
Subfields: Cancer Research, Immunology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/e42345c594cc2c38f864d6d19d1a5a1f5e20ca8c
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Citations in the corpus
- Cellular response to oxidative stress: Signaling for suicide and survival * (2002, result): “However, more recent studies have failed to provide support for this hypothesis as reviewed in Li and Karin (1999) and Bowie and O’Neill (2000).”
- iNOS-mediated nitric oxide production and its regulation. (2004, background): “In addition, cell-produced NO can block heme incorporation into iNOS monomer although this inhibition by NO is reduced in the presence of NO scavengers, O2 or superoxide (Bowie and O’Neill, 2000).”
- Reactive oxygen species in cell signaling. (2000, background): “Recent reviews (38, 167) on this subject have emphasized the importance of the recognition that a redox-dependent activation of NF-kB is cell and stimulus specific as opposed to the concept that oxidative stress is a common mediator of diverse NF-kB activators.”