Selective Vulnerability of Late Oligodendrocyte Progenitors to Hypoxia–Ischemia
By S. Back, B. Han, N. Luo, Charlene A Chricton, S. Xanthoudakis, J. Tam, Kara L. Arvin, D. Holtzman. Journal of Neuroscience, 2002
791 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Pediatrics, Perinatology and Child Health, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/e2feed2f4e580abdfe0513af7e231d9cd95b29ce
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In Endocrinology, Diabetes and Metabolism
In Pediatrics, Perinatology and Child Health
In Pulmonary and Respiratory Medicine
Citations in the corpus
- White matter changes in Alzheimer’s disease: a focus on myelin and oligodendrocytes (2018, positive): “Late-stage oligodendrocyte progenitors are more sensitive to hypoxic or ischemic damage than early-stage progenitors and more mature oligodendrocytes [3].”
- Dysregulation of the Wnt pathway inhibits timely myelination and remyelination in the mammalian CNS. (2009, background): “timely myelination and remyelination in the mammalian CNS”
- Neurodevelopmental and growth impairment among extremely low-birth-weight infants with neonatal infection. (2004, neutral): “Weight 10th percentile 1070/2138 (50) 494/915 (54)† 125/275 (46) 104/195 (53) 53/104 (51) 223/393 (57)†”
- The pathobiology of vascular dementia (2013, positive): “First, OPC in the late stage of development are particularly susceptible to injury in conditions of chronic hypoxia and oxidative stress existing in the ischemic white matter (Back et al., 2011; 2002; Fernando et al., 2006; French et al., 2009).”