Brain abnormalities in neuromyelitis optica.

By S. Pittock, V. Lennon, K. Krecke, D. Wingerchuk, C. Lucchinetti, B. Weinshenker. Archives of Neurology, 2006

698 citations recorded by the source corpus.

Subfields: Pathology and Forensic Medicine, Physiology, Surgery

View Paper: https://www.semanticscholar.org/paper/d818bd6ac71130b7105d6d70c2f769153d5c1d2b

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Pathology and Forensic Medicine

1. Hans Lassmann
2. C. Lucchinetti
3. B. Weinshenker
4. D. Wingerchuk
5. S. Pittock
6. V. Lennon
7. Wolfgang Brück

In Physiology

1. Joseph E. Parisi
2. E. Benarroch

Citations in the corpus

• Differential diagnosis of suspected multiple sclerosis: a consensus approach (2008, neutral): “medulla, and other brainstem areas [41,42].”
• Anti-aquaporin-4 antibody is involved in the pathogenesis of NMO: a study on antibody titre. (2007, background): “Consistent with the distribution of AQP4, the brain lesions of NMO were reported to be localized in the periventricular and periaqueductal areas (Misu et al., 2005a; Nakashima et al., 2006; Pittock et al., 2006a, b), and myelitis in NMO often involves the central gray matter.”
• Neuromyelitis optica and non organ-specific autoimmunity. (2008, background): “%) of the 18 patients with NMO or who were at risk for NMO.”
• Pattern-specific loss of aquaporin-4 immunoreactivity distinguishes neuromyelitis optica from multiple sclerosis. (2007, background): “Recent MRI studies have established conclusively that lesions in NMO may target the brain, even relatively early in the course of the disease, and that certain brain lesions are far more common in NMO than in MS (Pittock et al., 2006a,b).”
• MOG-IgG in NMO and related disorders: a multicenter study of 50 patients. Part 2: Epidemiology, clinical presentation, radiological and laboratory features, treatment responses, and long-term outcome (2016, background): “5 months since onset, median 52 [1-507] months), the disease took a relapsing course in most cases.”
• Loss of aquaporin 4 in lesions of neuromyelitis optica: distinction from multiple sclerosis. (2007, background): “Interestingly, the distribution of AQP4-rich areas in the central nervous system, such as the hypothalamus or the grey matter of the spinal cord, is compatible with that of NMO lesions (Misu et al., 2005; Nakashima et al., 2006; Pittock et al., 2006).”