Pattern-specific loss of aquaporin-4 immunoreactivity distinguishes neuromyelitis optica from multiple sclerosis.
By S. Roemer, Joseph E. Parisi, V. Lennon, E. Benarroch, Hans Lassmann, W. Bruck, R. Mandler, B. Weinshenker, S. Pittock, D. Wingerchuk and 1 more. Brain : a journal of neurology, 2007
704 citations recorded by the source corpus.
Subfields: Molecular Biology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/b969b69630e075bd3b444063ddd5beabd58bd3ae
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- Toshiyuki Takahashi
- K. Fujihara
- I. Nakashima
- T. Misu
- I. Miyazawa
- Masashi Nakamura
- Y. Itoyama
- Y. Shiga
Citations in the corpus
- Anti-aquaporin-4 antibody is involved in the pathogenesis of NMO: a study on antibody titre. (2007, background): “We and others most recently found the loss of AQP4 in the active perivascular lesions of NMO (Misu et al., 2006, 2007; Roemer et al., 2007).”