Loss of aquaporin 4 in lesions of neuromyelitis optica: distinction from multiple sclerosis.

By T. Misu, K. Fujihara, A. Kakita, H. Konno, M. Nakamura, S. Watanabe, Tetsuya Takahashi, I. Nakashima, Hitoshi Takahashi, Y. Itoyama. Brain : a journal of neurology, 2007

617 citations recorded by the source corpus.

Subfields: Atomic and Molecular Physics, and Optics

View Paper: https://www.semanticscholar.org/paper/3178f2840f5209365f4761920299312a41f3bc35

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

1. Toshiyuki Takahashi
2. K. Fujihara
3. I. Nakashima
4. T. Misu
5. I. Miyazawa
6. Masashi Nakamura
7. Y. Itoyama
8. Y. Shiga

Citations in the corpus

• Anti-aquaporin-4 antibody is involved in the pathogenesis of NMO: a study on antibody titre. (2007, background): “We and others most recently found the loss of AQP4 in the active perivascular lesions of NMO (Misu et al., 2006, 2007; Roemer et al., 2007).”
• Physiological roles of aquaporin-4 in brain. (2013, neutral): “There is an extensive literature on the role of AQP4 in neuromyelitis optica (NMO) (25, 26, 62, 70, 84, 106, 145, 146, 152, 153, 160).”