Subcutaneous and visceral adipose tissue: their relation to the metabolic syndrome.

By B. Wajchenberg. Endocrine reviews, 2000

3,056 citations recorded by the source corpus.

Subfields: Endocrine and Autonomic Systems, Epidemiology, Physiology

View Paper: https://www.semanticscholar.org/paper/d5e31cf35591eb95fc183a6776e25f8dab82a6b3

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In Endocrine and Autonomic Systems

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Citations in the corpus

The role of estrogens in control of energy balance and glucose homeostasis. (2013, background): “Indeed, intra-abdominal adipose tissue has more capillaries and efferent sympathetic axons per unit volume than sc fat, and unlike sc fat, it drains into the hepatic portal vein (121).”
Association of sleep time with diabetes mellitus and impaired glucose tolerance. (2005, methodology): “Waist girth was used to measure obesity, because it correlates better than either BMI or waist-hip ratio with visceral adiposity,(21) which is more relevant than total body fat to the pathophysiological features of DM.(22,23) In contrast to the Nurses’ Health Study report,(17) adjustment for obesity”
Overweight, obesity and cancer: epidemiological evidence and proposed mechanisms (2004, background): “Increased release of free fatty acids, resistin and TNFα by adipose tissue and reduced release of adiponectin give rise to insulin resistance — a metabolic state characterized by reduced metabolic response of tissues (muscle, liver, adipose) to insulin — and to compensatory hyperinsulinaemi”
Visceral Fat Is an Independent Predictor of All‐cause Mortality in Men (2006, background): “termed the Portal Theory (25), excess release of proinflammatory and prothrombotic factors may be the mechanism that links visceral fat and metabolic risk (26,27).”
Chemerin is a novel adipokine associated with obesity and metabolic syndrome. (2007, background): “Cross-sectional studies in humans suggest that levels of visceral adipose tissue are more strongly associated with insulin resistance than levels of sc tissue (15).”
The Pathophysiology of Gestational Diabetes Mellitus (2018, background): “Non-diabetic obese individuals exhibit adequate adipose tissue expansion in response to fuel surfeit, and therefore maintain healthy blood glucose concentrations, sufficient β-cell compensation, and avoid chronic insulin resistance [110,111].”
beta-cell failure in diabetes and preservation by clinical treatment. (2007, background): “Regarding GLP-1, DPP-IV activation results in the inactivation of GLP-1 (7-36) amide and the generation of the metabolite GLP-1 (9-36) amide, which did not activate the GLP-1 receptor, thus not enhancing the insulin secretion and not blocking GLP-1 (7-36) amide enhancement of insulin secretion durin”
Adipocyte Turnover: Relevance to Human Adipose Tissue Morphology (2009, background): “Although the amount and distribution of adipose tissue associate independently with insulin resistance, type 2 diabetes, and other metabolic disorders (1), the size of adipocytes within the adipose tissue is also important (2).”