ACF catalyses chromatosome movements in chromatin fibres

By V. Maier, Mariacristina Chioda, D. Rhodes, Peter B. Becker. 2008

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The genomic landscape of the somatic linker histone subtypes H1.1 to H1.5 in human cells. (2013, background): “Moreover, it was recently demonstrated that in vitro, chromatin-con- 2142 Cell Reports 3, 2142–2154, June 27, 2013 ª2013 The Authors taining H1 is not completely inaccessible and can still be remodeled by ATPase-containing complexes (Maier et al., 2008; Clausell et al., 2009).”
Gene-specific H1 eviction through a transcriptional activator-p300-NAP1-H1 pathway (2019, background): “, 2002) of chromatin templates, as well as in nucleosome movements in H1-containing nucleosome arrays (Maier et al., 2008), was found to release H3/H4 (Fig.”
The biology of chromatin remodeling complexes. (2009, background): “Interestingly, dACF effectively slides chromatosomes, whereas dCHD1 cannot (61), a selectivity consistent with ISWI promoting H1 assembly into chromatin in vivo (57, 62).”
Cell-free genomics reveal intrinsic, cooperative and competitive determinants of chromatin interactions (2021, result): “This is in line with biochemical observations that ISWI can slide chromatosomes (78,79) This situation may well reflect the in vivo situation, since ISWI remodelers are very abundant (80).”
The ISW1 and CHD1 ATP-dependent chromatin remodelers compete to set nucleosome spacing in vivo (2016, background): “Remodeling of nucleosomal arrays by dCHD1 is inhibited by H1 (60), potentially explaining the dominance of ISW1 in the competition with CHD1.”