Endocannabinoid levels in rat limbic forebrain and hypothalamus in relation to fasting, feeding and satiation: stimulation of eating by 2‐arachidonoyl glycerol

By T. Kirkham, C. Williams, F. Fezza, V. Di Marzo. British Journal of Pharmacology, 2002

801 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Endocrine and Autonomic Systems, Pharmacology

View Paper: https://www.semanticscholar.org/paper/cfe423b282c570491556707ccd679ca1f065b7c6

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cognitive Neuroscience

1. U. Kumar
2. Shenglong Zou
3. George Kunos
4. V. Di Marzo

In Endocrine and Autonomic Systems

1. V. Di Marzo

In Pharmacology

1. V. Di Marzo
2. U. Kumar
3. Shenglong Zou
4. George Kunos
5. P. Pacher
6. S. Batkai
7. G. Marsicano
8. B. Lutz

Citations in the corpus

• The Endocannabinoid System as an Emerging Target of Pharmacotherapy (2006, background): “Similar increases in food intake can be elicited by 2-AG administered systemically or into the nucleus accumbens shell region (Kirkham et al., 2002) or into the lateral hypothalamus (Kirkham and Williams, 2001a).”
• The emerging role of the endocannabinoid system in endocrine regulation and energy balance. (2006, background): “Interestingly, unchanged levels of endocannabinoids were shown in the cerebellum, a region not involved in the control of feeding, further confirming the notion that endocannabinoids are produced in situ and on demand (264).”
• Regulation, function, and dysregulation of endocannabinoids in models of adipose and beta-pancreatic cells and in obesity and hyperglycemia. (2006, methodology): “• Endocannabinoids in Obesity J Clin Endocrinol Metab, August 2006, 91(8):3171–3180 3177”
• Cannabinoid Receptors and the Endocannabinoid System: Signaling and Function in the Central Nervous System (2018, background): “The stimulation of appetite and feeding behavior is observed after direct injection of endocannabinoids and is abolished by the administration of CB1R antagonists [167].”
• Targeting the endocannabinoid system: to enhance or reduce? (2008, background): “In rodents, hypothalamic endocannabinoid (particularly 2-AG) levels first increase after ∼18 hours food deprivation and then decrease after food consumptio”
• Nongenomic Glucocorticoid Inhibition via Endocannabinoid Release in the Hypothalamus: A Fast Feedback Mechanism (2003, background): “Glucocorticoids and endocannabinoids both have well established central orexigenic actions (Zakrzewska et al., 1999; Kirkham et al., 2002) such that the modulatory cross talk between glucocorticoids and endocannabinoids that leads to reduced excitation of PVN parvocellular neurons demonstrated here ”