[Pathophysiology of coronary artery disease].

By C. Bauters. La Revue du praticien, 2008

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Update on acute coronary syndromes: the pathologists' view. (2013, background): “Coronary heart disease is nearly always caused by coronary atherosclerosis with or without luminal thrombosis and vasospasm.(4,5) Atherosclerosis alone may cause stable angina but is rarely fatal.”
Cytokines in atherosclerosis: pathogenic and regulatory pathways. (2006, background): “It also acts at the onset of adverse clinical vascular events, when activated cells within the plaque secrete matrix proteases that degrade extracellular matrix proteins and fragilize the fibrous cap, leading to rupture and thrombus formation (399).”
The Endocannabinoid System as an Emerging Target of Pharmacotherapy (2006, background): “Atherosclerosis—Chronic inflammation and the associated oxidative-nitrosative stress are key players in atherosclerosis and cardiovascular aging, and pharmacological modulation of these processes could be of therapeutic benefit (reviewed in Csiszar et al., 2005; Libby and Theroux, 2005).”
Development and validation of improved algorithms for the assessment of global cardiovascular risk in women: the Reynolds Risk Score. (2007, background): “Furthermore, over the past halfcentury, understanding of the biological processes underlying atherothrombosis has markedly shifted to encompass the complex biology of hemostasis, thrombosis, inflammation, endothelial dysfunction, and plaque instability.(11,12) Despite this changing view of pathophys”