Signalling: STATs: transcriptional control and biological impact

By D. Levy, James E. Darnell. Nature reviews. Molecular cell biology, 2002

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Dangerous liaisons: STAT3 and NF-κB collaboration and crosstalk in cancer (2009, background): “Other STAT3 partners include androgen receptor, C/EBP, STAT6, STAT1, c-Jun, and β-catenin (63).”
gp130-mediated Stat3 activation in enterocytes regulates cell survival and cell-cycle progression during colitis-associated tumorigenesis. (2009, background): “Persistent and aberrant activation of various members of the signal transducer and activator of transcription (STAT) family is a recurrent and unifying molecular feature of many human malignancies of hematological and epithelial origin (Levy and Darnell, 2002; Yu and Jove, 2004).”
The molecular control of corpus luteum formation, function, and regression. (2007, background): “A single nucleotide difference between Stat5 response elements located on the ER and ER promoters is responsible for the observation that either Stat5a or Stat5b can stimulate ER transcription, whereas only Stat5b can activate transcription of ER (83).”
STAT3 signaling in immunity (2016, background): “signaling mechanisms and functions in the early days of the field [37–42].”