The molecular basis of skeletal muscle atrophy.
By R. Jackman, S. Kandarian. American Journal of Physiology - Cell Physiology, 2004
991 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Genetics, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/c967fdcdc7c19423a0777b702d33fa16f65c2c35
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In Cardiology and Cardiovascular Medicine
In Genetics
In Molecular Biology
- A. Goldberg
- D. Glass
- L. Valente
- J. Fernandes
- I. Johnston
- Steven P. Gygi
- Luís E. C. Conceição
- J. Brault
Citations in the corpus
- Plant protein blends in diets for Senegalese sole affect skeletal muscle growth, flesh texture and the expression of related genes (2016, background): “Most studies evaluated the impact of such diets on the nutritional value of the fillet, but few focused on organoleptic attributes and even less on textural properties (de Francesco et al., 2004; Johnsen et al., 2011; Matos et al., 2012).”
- What determines growth potential and juvenile quality of farmed fish species (2013, background): “Protein degradation can be mediated by four main proteolytic systems: calpains, caspases, lysosomes and the ubiquitin-proteasome system (UPS; Jackman & Kandarian 2004).”
- During muscle atrophy, thick, but not thin, filament components are degraded by MuRF1-dependent ubiquitylation (2009, background): “Atrophy of skeletal muscle occurs upon disuse or denervation and systemically in fasting and many disease states, including cancer cachexia, sepsis, renal failure, and cardiac failure (Lecker et al., 1999, 2006; Jackman and Kandarian, 2004).”