Oxytocin increases retention of social cognition in autism.

By E. Hollander, Jennifer A. Bartz, W. Chaplin, Ann T. Phillips, Jennifer M Sumner, L. Soorya, E. Anagnostou, S. Wasserman. Biological Psychiatry, 2007

732 citations recorded by the source corpus.

Subfields: Behavioral Neuroscience, Cognitive Neuroscience, Social Psychology

View Paper: https://www.semanticscholar.org/paper/c5cb6503d379c0b0d9b14ecd339decdaa6dbd48b

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Citations in the corpus

The Challenge of Translation in Social Neuroscience: A Review of Oxytocin, Vasopressin, and Affiliative Behavior (2010, result): “(Andari et al.; Guastella et al., 2009a; Hollander et al., 2007; Hollander et al., 2003) While not a cure, the results are promising.”
Oxytocin and the neural mechanisms regulating social cognition and affiliative behavior. (2009, background): “A more important finding of this study was that prior exposure to OT increased the learning ability to subsequent emotional tasks [110].”
Oxytocin and vasopressin in the human brain: social neuropeptides for translational medicine (2011, background): “; however, understanding inter-individual variation in the OXT system is important considering the current interest in treating ASD with OX”
Consensus Paper: Pathological Role of the Cerebellum in Autism (2012, background): “Improvements in repetitive behaviors in adults with autism following oxytocin infusion have been demonstrated [169] as well as positive effects on social behaviors and cognition [170].”
Oxytocin: the Great Facilitator of Life (2009, unknown): “…disorder significantly reduces both number and severity of repetitive behaviors (such as repeating, self-injury, touching; Hollander et al., 2003), and increases ability to comprehend and remember the affective component of spoken words (happy, indifferent, angry, or sad (Hollander et al., 2007)).”
A common allele in the oxytocin receptor gene (OXTR) impacts prosocial temperament and human hypothalamic-limbic structure and function (2010, background): “Though this outcome is well in line with the presumed role of OT function in human sociality, its pathophysiological significance remains to be elucidated, as evidence for a hypothalamus dysfunction in social disorders such as autism is hitherto limited to single case studies (32, 55) and indirect m”
Early behavioral intervention, brain plasticity, and the prevention of autism spectrum disorder (2008, background): “meaning (Hollander et al., 2007) in individuals with ASD.”
Promoting social behavior with oxytocin in high-functioning autism spectrum disorders (2010, background): “Although previous studies have shown that oxytocin can reduce repetitive behavior in subjects with autism (28) and enhance the comprehension of affective speech (29), here we demonstrated that oxytocin can promote social approach and social comprehension in patients with autism.”
The Social Salience Hypothesis of Oxytocin. (2016, unknown): “led researchers to evaluate its therapeutic applications in various psychiatric conditions such as autism, schizophrenia and personality disorder, yielding mixed results (35-40).”
Close social associations in animals and humans : functions and mechanisms of friendship (2010, unknown): “, happy, sad) (Hollander et al., 2007).”
Developing constructs for psychopathology research: research domain criteria. (2010, unknown): “, Kosfeld, Heinrichs, Zak, Fischbacher, & Fehr, 2005; Thompson, Gupta, Miller, Mills, & Orr, 2004), including some work pointing to potential therapeutic effects for specific disorders (Hollander et al., 2007; Insel, 1999).”
Brain basis of human social interaction: from concepts to brain imaging. (2009, background): “Oxytocin improves a person’s ability to infer mental states from social cues in the eye region (47), and, interestingly, some preliminary experiments have already been made to show that oxytocin administration can be beneficial for treatment of autistic persons; after oxytocin treatment, adult autis”