Role of body fat distribution and the metabolic complications of obesity.

By M. Jensen. Journal of Clinical Endocrinology and Metabolism, 2008

697 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Endocrinology, Diabetes and Metabolism, Epidemiology

View Paper: https://www.semanticscholar.org/paper/4b2d46f4fbdfafb51ae8253c6fa8344b2ff9b8bf

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. André Tchernof
2. Ralph DeFronzo
3. J. Despres
4. Gerald I. Shulman
5. Melania Gaggini
6. Amalia Gastaldelli
7. Emma Buzzigoli
8. S. Friedman

In Endocrinology, Diabetes and Metabolism

1. André Tchernof
2. Gerald I. Shulman
3. V. Samuel
4. J. Despres
5. S. Friedman
6. Ralph DeFronzo
7. Melania Gaggini
8. Amalia Gastaldelli

In Epidemiology

1. André Tchernof
2. V. Samuel
3. Gerald I. Shulman
4. S. Friedman
5. Ralph DeFronzo
6. J. Despres
7. Melania Gaggini
8. Amalia Gastaldelli

Citations in the corpus

• Sex differences in human adipose tissues – the biology of pear shape (2012, background): “These so-called upper body obese women suffer from the same metabolic complications as men [92].”
• Mechanisms and disease consequences of nonalcoholic fatty liver disease (2021, background): “However, it is also possible that VAT simply serves as another ectopic site for lipids when subcutaneous adipose tissue capacity is exceeded (Cuthbertson et al., 2017; Jensen, 2008; Smith, 2015) and would therefore not play a primary causal role in metabolic dysregula-”
• Pathophysiology of human visceral obesity: an update. (2013, background): “Such robust evidence has also been previously detailed in several review papers on body fat topography published over the last 20 years (48, 71, 72, 112, 113, 234, 263, 357, 360).”
• Transcriptional control of adipose lipid handling by IRF4 (2011, neutral): “The current epidemic of obesity has prompted intense investigation into the molecular mechanisms underlying fat storage and utilization (Bluher, 2009; Jensen, 2008).”
• Non-Alcoholic Fatty Liver Disease (NAFLD) and Its Connection with Insulin Resistance, Dyslipidemia, Atherosclerosis and Coronary Heart Disease (2013, positive): “It is well established that elevated free fatty acids (FFA) stimulate hepatic GNG [47] and VLDL-TG production in the face of hyperinsulinemia [40].”
• Obesity and Cancer: The Role of Dysfunctional Adipose Tissue (2009, background): “Abdominal adipose tissue, which is strategically located to the liver, is especially associated with an abnormal metabolic profile (37).”
• Non-Alcoholic Fatty Liver Disease as a Nexus of Metabolic and Hepatic Diseases (2017, positive): “However, many now consider VAT as another ectopic site for lipids when subcutaneous adipose tissue capacity is exceeded (Cuthbertson et al., 2017; Jensen, 2008; Smith, 2015).”